Author

Miriam Eckstein

University of WürzburgORCID 0000-0003-2847-9044Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Katrin SinningiD· 2 shared Martin VaethiD· 2 shared Xiufeng ZhaoiD· 2 shared Anke FreitagiD· 1 shared Christian StigloheriD· 1 shared Christoph MaackiD· 1 shared Hao WuiD· 1 shared Jan DudekiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersImmunology

Katrin SinningORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

ImmunologyEuropean Journal of ImmunologyPeer-reviewedAug 1, 2026

The Glucose Transporter GLUT3 Controls Regulatory T Cell Function

Katrin Sinning, Miriam Eckstein, Xiufeng Zhao…·Martin Vaeth

This study shows that GLUT3 is essential for Treg cell function and immune regulation, challenging the idea that Treg cells rely mainly on mitochondrial metabolism.

DOI 10.1002/eji.70272

mouseproteomics

𝕏 PostBluesky

ImmunologybioRxivPreprintAug 25, 2025

Tafazzin-Mediated Cardiolipin Remodeling Controls Metabolic Stress Response and Effector Function of Inflammatory T Cells

Xiufeng Zhao, Sophia M. Hochrein, Miriam Eckstein…·Martin Vaeth

This study shows that Tafazzin-mediated cardiolipin remodeling is essential for metabolic stress response and effector function of inflammatory T cells, with implications for Barth syndrome.

DOI 10.1101/2025.08.25.672061

humanmouse

𝕏 PostBluesky