Katrin Sinning

This study shows that GLUT3 is essential for Treg cell function and immune regulation, challenging the idea that Treg cells rely mainly on mitochondrial metabolism.

This study shows that Tafazzin-mediated cardiolipin remodeling is essential for metabolic stress response and effector function of inflammatory T cells, with implications for Barth syndrome.

This study shows that glucose uptake via GLUT1/3 is essential for neutrophil oxidative burst and lipid mediator production during microbial infection.

This study uses single-cell RNA-sequencing and spatial transcriptomics to analyze the spatio-temporal dynamics of the fibrotic niche in cardiac repair after ventricular injury in adult mice, identifying key cell interactions and regulatory mechanisms.