Author

David Wu

University of ChicagoORCID 0000-0003-3162-3238Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

David Wu1 paper · University of Michigan

Biology Disciplines

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Bioengineering· 1 Genomics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Organism:mouse· 2 Assay:ATAC-seq· 1

Frequent Co-Authors

Yun Hua FangiD· 3 shared Gökhan M. MutluiD· 2 shared Ru‐Ting HuangiD· 2 shared Tzu‐Pin ShentuiD· 2 shared Aaron T. AlpariD· 1 shared Ada WeinstockiD· 1 shared Aleepta Guha RayiD· 1 shared Angelo Y. Meliton· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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3 shared papersBioengineering

Yun Hua FangORCID

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3 papers

Recent·Most cited

BioengineeringbioRxivPreprintJan 9, 2025

Precision mRNA Nanomedicine for Targeted Vascular Therapies in ARDS and Atherosclerosis

Zhengjie Zhou, Jiayu Zhu, Chih‐Fan Yeh…·Yun Hua Fang

This paper develops a modular nanoparticle platform for targeted mRNA delivery to inflamed endothelial cells, showing efficacy in treating ARDS and atherosclerosis by restoring disease-related gene pathways.

6 citations

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Molecular BiologybioRxivPreprintApr 29, 2021

Decitabine Reactivation of FoxM1-Dependent Endothelial Regeneration and Vascular Repair for Potential Treatment of Elderly ARDS and COVID-19 Patients

Xiaojia Huang, Xianming Zhang, Narsa Machireddy…·You‐Yang Zhao

This study shows that Decitabine can reactivate FoxM1-dependent vascular repair in aged mice, improving survival after sepsis and suggesting potential treatment for elderly ARDS and COVID-19 patients.

3 citations

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GenomicsProceedings of the National Academy of SciencesPeer-reviewedNov 14, 2018

A genetic variant at coronary artery disease and ischemic stroke locus 1p32.2 regulates endothelial responses to hemodynamics

Matthew D. Krause, Ru‐Ting Huang, David Wu…·Yun Hua Fang

This study identifies a genetic variant at 1p32.2 that regulates endothelial responses to hemodynamics by modulating an enhancer region in vascular endothelium.

75 citationstop 5%

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