Author

Wendy R. Kates

SUNY Upstate Medical UniversityORCID 0000-0003-3358-2572Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:imaging· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Carrie E. BeardeniD· 3 shared Declan MurphyiD· 3 shared Charles H. SchleiferiD· 2 shared Leila KushaniD· 2 shared A. Elizabeth de GuzmaniD· 1 shared Alan AnticeviciD· 1 shared Alberto GalbuseraiD· 1 shared Alessandro GozziiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersNeuroscience

Carrie E. BeardenORCID

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3 papers

Recent·Most cited

NeuroscienceScience AdvancesPeer-reviewedMar 12, 2025

Synaptic-dependent developmental dysconnectivity in 22q11.2 deletion syndrome

Filomena Grazia Alvino, Silvia Gini, Antea Minetti…·Alessandro Gozzi

This study investigates developmental dysconnectivity in 22q11.2 deletion syndrome using a cross-species approach, finding age-specific fMRI connectivity changes with synaptic origins.

28 citationstop 1%

mousehuman

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NeuroscienceBiological PsychiatryPeer-reviewedAug 23, 2024

Unique functional neuroimaging signatures of genetic versus clinical high risk for psychosis

Charles H. Schleifer, Sarah E. Chang, Carolyn Amir…·Carrie E. Bearden

The paper compares functional brain measures between 22qDel and CHR cohorts, finding distinct patterns of connectivity and variability disruptions.

7 citations

humanimaging

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GeneticsbioRxivPreprintSep 30, 2022

Chromatin regulators in the TBX1 network confer risk for conotruncal heart defects in 22q11.2DS and sporadic congenital heart disease

Yingjie Zhao, Yujue Wang, Lijie Shi…·Bernice E. Morrow

The paper identifies chromatin regulatory genes as genetic modifiers of conotruncal heart defects in 22q11.2 deletion syndrome and links them to sporadic congenital heart disease.

DOI 10.1101/2022.09.30.507111

mousewhole-genome sequencing

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