Alberto Galbusera

This study shows that endogenous oxytocin alters brain network dynamics, increasing salience-related network states and decreasing sensory-dominated states in mice.

The paper shows that cortical excitability inversely affects fMRI connectivity, with low-frequency neuronal coupling being the key driver of these changes.

This paper establishes a transcranial fUSI protocol and preprocessing framework to map mouse brain networks, showing high spatial concordance with fMRI and revealing structure-function coupling and dynamic resting-state patterns.

This study shows that a transient developmental excitation-inhibition imbalance causes lasting social brain circuit dysfunction in mice, with behavioral and transcriptional consequences.

This study investigates developmental dysconnectivity in 22q11.2 deletion syndrome using a cross-species approach, finding age-specific fMRI connectivity changes with synaptic origins.

The paper identifies hypo- and hyperconnectivity subtypes in autism using cross-species fMRI, linking them to distinct biological mechanisms in mice and humans.

This study shows that Ube3a gene overdosage causes sex-biased effects on brain connectivity, behavior, and gene expression in mice, linking it to autism-related phenotypes.

This paper investigates brain network and behavioral changes in a mouse model of CTD and shows that perinatal gene therapy can partially reverse some symptoms but not all.

This study shows that complement signaling via C3r is critical for perinatal neuronal elimination but not synaptic pruning in the developing brain.

The paper shows that silencing the prefrontal cortex paradoxically increases resting-state fMRI connectivity with its thalamo-cortical terminals.

This study investigates spatiotemporal dynamics of spontaneous brain activity in awake mice using resting-state fMRI, finding unique patterns distinct from anesthetized states.

The paper links mTOR-related synaptic pathology to functional hyperconnectivity in autism using mouse models and human fMRI data.

This study investigates somatosensory processing deficits in Shank3b-/- mice, finding reduced cortical and hippocampal activation and connectivity linked to hypo-responsiveness to tactile stimuli.

This study investigates how xanomeline, an M1/M4 agonist, affects brain functional connectivity in mice using phMRI, rsfMRI, and qEEG, showing both direct and modulatory effects on neural activity and connectivity.

The paper maps brain connectivity in 16 autism mouse models to identify four subtypes of functional connectivity abnormalities linked to different etiologies.

This study examines how acute and repeated intranasal oxytocin administration affects brain-wide functional connectivity in mice, revealing distinct patterns of neural reconfiguration with potential clinical implications.

This study shows that deleting the autism risk gene Shank3 in mice disrupts prefrontal connectivity and is linked to social communication deficits.

This study investigates how serotonin deficiency affects hippocampal neuroplasticity and emotional behavior in mice, revealing a buffering role of serotonin in adaptive responses to environmental stress.

This study examines Chd8 haploinsufficient mice, finding brain overgrowth, altered gene expression, and functional connectivity changes linked to autism-like features.

This study uses chemogenetic-fMRI to identify brain regions activated by serotonergic stimulation, showing region-specific effects rather than global activity changes.

This study shows that homozygous loss of CNTNAP2 in mice reduces prefrontal and midline brain functional connectivity, linking it to autism-related traits.