Brady L. Spencer

This study shows that GBS infection increases arachnoid barrier permeability in neonatal mice, driven by cytokine production from fibroblasts and macrophages.

This study shows that collagen binding adhesin (Cna) in Staphylococcus aureus reduces skin infection severity by binding to C1q and limiting inflammation.

The paper identifies glycolipids and lipoteichoic acid alanylation as factors contributing to Group B streptococcal diabetic wound infection using a transposon mutant screen in a murine model.

This study investigates the cellular immune response to GBS vaginal colonization in mice, finding that neutrophils and γδ T cells contribute to host defense, while overall immune cell numbers remain stable.

This study identifies Glyoxalase A (gloA) in Group B Streptococcus as essential for methylglyoxal tolerance and virulence during infection.

This study identifies a GBS T7SS-secreted toxin, BltA, that mediates interbacterial competition and vaginal colonization by inhibiting Enterococcus faecalis in vitro and in vivo.