Ben J. Vincent

The paper investigates the regulatory landscape of female and male genital structures in Drosophila melanogaster, finding shared transcription factors and their expression patterns in both sexes.

The paper defines the kinetic roles of three transcription factors in the Drosophila embryo by analyzing their effects on transcriptional output through live imaging.

The paper shows that a mutation in a minimal enhancer of Drosophila eve stripe 2 is buffered by flanking sequences in extended enhancer constructs, but not in the endogenous context.

The paper maps transcription factor expression in Drosophila melanogaster pupal terminalia using RNA-seq and in situ hybridization, revealing structured patterns and creating an online database for future research.

The paper investigates the HbP2 enhancer in Drosophila, showing that cooperative binding of Bcd is insufficient to explain its sharp expression pattern, and identifies other factors like pioneer factors and histone modifiers as key contributors.

The paper shows that Caudal counter-represses Hunchback to enable even-skipped stripe 2 expression in Drosophila embryos, challenging the traditional view of Hunchback as a direct activator.

The paper shows that Hunchback regulates two enhancers with opposite activities to produce the same gene expression pattern in Drosophila embryos.