Author

Min K. Tsai

Stanford MedicineORCID 0000-0003-4732-4259Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Dmitri A. PetroviD· 2 shared Emily G. ShuldineriD· 2 shared Hongchen CaiiD· 2 shared Laura AndrejkaiD· 2 shared Monte M. WinslowiD· 2 shared Alberto Diaz-JimeneziD· 1 shared Benedikt BrorsiD· 1 shared Christian A. KunderiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCancer Biology

Dmitri A. PetrovORCID

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2 papers

Recent·Most cited

Cancer BiologyNature AgingPeer-reviewedNov 4, 2025

Aging represses lung tumorigenesis and alters tumor suppression

Emily G. Shuldiner, Saswati Karmakar, Min K. Tsai…·Monte M. Winslow

This study shows aging suppresses lung tumor initiation and growth in mice, with age-dependent effects on tumor suppressor genes like PTEN and persistent age-related transcriptomic changes in tumors.

16 citations

mousescRNA-seq

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Cancer BiologyCancer DiscoveryPeer-reviewedSep 23, 2025

EML4-ALK variant-specific genetic interactions shape lung tumorigenesis

Alberto Diaz-Jimenez, Emily G. Shuldiner, Kálmán Somogyi…·Rocı́o Sotillo

The paper investigates functional differences between EML4-ALK variants V1 and V3 in lung cancer using mouse models and human data, showing variant-specific effects on tumorigenesis.

8 citationstop 4%

mouseDNA methylation

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