Author

Paolo Bellotti

University of Florida HealthORCID 0000-0003-4839-8807Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

Gilbert R. UpchurchiD· 3 shared Jonathan R. KrebsiD· 3 shared Shiven SharmaiD· 3 shared Adithan AravinthaniD· 2 shared Ashish Kumar SharmaiD· 2 shared Denny Joseph Manual KollarethiD· 2 shared Gang SuiD· 2 shared Guoshuai CaiiD· 2 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersImmunology

Gilbert R. UpchurchORCID

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintJul 7, 2026

YAP/TAZ Signaling in Endothelial Cells Mediates the Pathogenesis of Abdominal Aortic Aneurysm Formation

Walker Ueland, Paolo Bellotti, Jeff Arni C. Valisno…·Gilbert R. Upchurch

This study identifies YAP/TAZ signaling in endothelial cells as a key mediator of abdominal aortic aneurysm formation and shows that pharmacological inhibition reduces AAA progression.

DOI 10.64898/2026.07.01.735919

humanmouse

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ImmunologyArteriosclerosis Thrombosis and Vascular BiologyPeer-reviewedOct 2, 2025

Pharmacologic Inhibition of Ferroptosis Attenuates Experimental Abdominal Aortic Aneurysm Formation

Jonathan R. Krebs, Paolo Bellotti, Walker Ueland…·Gilbert R. Upchurch

This study shows that inhibiting ferroptosis with liproxstatin-1 reduces AAA formation by decreasing inflammation and aortic remodeling in mice.

16 citationstop 1%

mousebulk RNA-seq

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ImmunologyThe FASEB JournalPeer-reviewedSep 25, 2024

Resolvin D2/GPR18 signaling enhances monocytic myeloid-derived suppressor cell function to mitigate abdominal aortic aneurysm formation

Paolo Bellotti, Zachary Ladd, Victoria Leroy…·Gilbert R. Upchurch

This study shows RvD2/GPR18 signaling enhances M-MDSC function to reduce AAA formation by suppressing inflammation and vascular remodeling.

14 citationstop 5%

mouseproteomics

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