Author

Tianshi David Wu

Baylor College of MedicineORCID 0000-0003-4906-3232Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

Alan WaichiD· 1 shared Armando M. MarrufoiD· 1 shared B.J. MossiD· 1 shared Benjamin A. RabyiD· 1 shared Charles G. Minard· 1 shared Chris H. WendtiD· 1 shared Cristian CoarfaiD· 1 shared David Brian CorryiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCell Biology

Alan WaichORCID

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2 papers

Recent·Most cited

A Mixed T2/T17-Associated Systemic Immune Signature Links Airborne Pollutant Exposure to Persistent Respiratory Symptoms

Armando M. Marrufo, Chris H. Wendt, Eric Garshick, Vincent S. Fan, Raúl San Jośe Estépar, Lizhen Song, Jiayi Li, Sarvika Periyapalayam Murali, Ivan M. Marrufo, Maxwell Stewart, Demerise Johnston, David Brian Corry, Tianshi David Wu, Farrah Kheradmand

The paper identifies a T2/T17 immune signature in individuals with persistent respiratory symptoms linked to airborne pollutant exposure and small-airway dysfunction.

bioRxiv · Aug 21, 2026 · PreprintImmunology

Single cell transcriptomics in a treatment-segregated cohort exposes a STAT3-regulated therapeutic gap in idiopathic pulmonary fibrosis

Neil J. McKenna, Scott A. Ochsner, Alan Waich, Juan Cala-García, Maria E. Ruiz Echartrea, Sandra L. Grimm, Fernando Poli, Rafael Cardenas Castillo, Juan D. Zuluaga, Sergio Poli, Taylor Sterling Adams, Ricardo Pineda, B.J. Moss, Tianshi David Wu, Charles G. Minard, , Iván O. Rosas

The paper uses single-cell transcriptomics to analyze an IPF cohort, revealing a transcriptional deficit refractory to current treatments and identifying STAT3 as a key regulator in the therapeutic gap.

bioRxiv · Jun 16, 2025 · PreprintCell Biology