Author

Ioanna Boumpoureka

Luxembourg Institute of HealthORCID 0009-0000-1795-6218Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Rejko KrügeriD· 2 shared Vyron A. GorgogietasiD· 2 shared Anne GrünewaldiD· 1 shared Arnaud PoretiD· 1 shared Elizabet PetkovskiiD· 1 shared Era TaoufikiD· 1 shared Felix Bruno Kleine BorgmanniD· 1 shared François MassartiD· 1 shared

Activity by Year

2022-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

Rejko KrügerORCID

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintOct 23, 2026

Generation of an induced pluripotent stem cell line, LCSBi021-A, from a Parkinson's disease patient harboring the missense mutation c.213C>G (p.Ser71Arg) in the RAB32 gene

Ioanna Boumpoureka, Vyron A. Gorgogietas, Elizabet Petkovski…·Rejko Krüger

The paper generates iPSCs from a Parkinson's patient with a RAB32 mutation and confirms their pluripotency and differentiation potential.

DOI 10.64898/2026.10.20.760406

humanbulk RNA-seq

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NeurosciencebioRxivPreprintSep 15, 2026

SNCA triplication shapes neuronal extracellular vesicle biology and promotes microglial activation in patient-derived iPSC-based models of Parkinson's disease

Ioanna Boumpoureka, Johanna Heider, Sylvie Delcambre…·Rejko Krüger

This study investigates how SNCA triplication affects neuronal extracellular vesicle biology and microglial activation in Parkinson's disease using iPSC-derived models.

DOI 10.64898/2026.09.09.750343

humanbulk RNA-seq

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Neurosciencenpj Parkinson s DiseasePeer-reviewedFeb 11, 2022

High Content Screening and Proteomic Analysis Identify a Kinase Inhibitor that rescuespathological phenotypes in a Patient-Derived Model of Parkinson's Disease

Nasia Antoniou, Kanella Prodromidou, Georgia Kouroupi…·Rebecca Matsas

The paper identifies a kinase inhibitor, BX795, that rescues pathological phenotypes in a patient-derived Parkinson's disease model through proteomic analysis and high-content screening.

40 citationstop 5%

humanproteomics

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