Author

Vyron A. Gorgogietas

Luxembourg Institute of HealthORCID 0000-0002-3211-6468Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 4 Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:imaging· 1

Frequent Co-Authors

Rejko KrügeriD· 4 shared Anne GrünewaldiD· 2 shared Ibrahim BoussaadiD· 2 shared Ioanna BoumpourekaiD· 2 shared Johannes H. WilbertziD· 2 shared Peter SommeriD· 2 shared Alessandro MichelucciiD· 1 shared Alexandre BaroniD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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4 shared papersNeuroscience

Rejko KrügerORCID

Mutual co-authors in cluster

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4 papers

Recent·Most cited

Cell BiologybioRxivPreprintOct 23, 2026

Generation of an induced pluripotent stem cell line, LCSBi021-A, from a Parkinson's disease patient harboring the missense mutation c.213C>G (p.Ser71Arg) in the RAB32 gene

Ioanna Boumpoureka, Vyron A. Gorgogietas, Elizabet Petkovski…·Rejko Krüger

The paper generates iPSCs from a Parkinson's patient with a RAB32 mutation and confirms their pluripotency and differentiation potential.

DOI 10.64898/2026.10.20.760406

humanbulk RNA-seq

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NeurosciencebioRxivPreprintSep 15, 2026

SNCA triplication shapes neuronal extracellular vesicle biology and promotes microglial activation in patient-derived iPSC-based models of Parkinson's disease

Ioanna Boumpoureka, Johanna Heider, Sylvie Delcambre…·Rejko Krüger

This study investigates how SNCA triplication affects neuronal extracellular vesicle biology and microglial activation in Parkinson's disease using iPSC-derived models.

DOI 10.64898/2026.09.09.750343

humanbulk RNA-seq

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NeuroscienceScientific ReportsPeer-reviewedAug 12, 2025

Morphological profiling reveals neuroprotection via mitochondrial uncoupling in human dopaminergic neurons

Vyron A. Gorgogietas, Amélie Weiss, Loïc Cousin…·Johannes H. Wilbertz

The paper identifies compounds that restore morphology in Parkinson's disease neurons by targeting mitochondrial uncoupling.

2 citations

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NeuroscienceBrainPeer-reviewedFeb 5, 2025

Parkinson's disease-related Miro1 mutation induces mitochondrial dysfunction and loss of dopaminergic neurons in vitro and in vivo

Axel Chemla, Giuseppe Arena, Ginevra Sacripanti…·Cláudia Saraiva

This study shows that a Miro1 mutation causes mitochondrial dysfunction and dopaminergic neuron loss in both cell models and mice, supporting its role in Parkinson's disease.

30 citationstop 1%

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