Author

Maud Tusseau

École Normale Supérieure de LyonORCID 0009-0001-2699-3827Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Genetics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

Alban C. ZiegleriD· 1 shared Alexandre BélotiD· 1 shared Alexandre GuilhemiD· 1 shared Alice LepelleyiD· 1 shared Antoinette PerlatiD· 1 shared Ariane GuillemotiD· 1 shared Aurélie GoyenvalleiD· 1 shared Béatrice Jaspard‐VinassaiD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperGenetics

Alban C. ZieglerORCID

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3 papers

Recent·Most cited

ImmunologyNature CommunicationsPeer-reviewedAug 19, 2026

NAP1 switches from an activator to a limiter of interferon induction by trapping TBK1 in condensates

Damien Glon, Quentin Riller, Lucı́a Álvarez…·Yves Gaudin

This study shows NAP1 switches from enhancing to limiting TBK1 activity through condensate formation, affecting IFN induction and linking to interferonopathies.

DOI 10.1038/s41467-026-76769-w

humanproteomics

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GeneticsbioRxivPreprintSep 13, 2024

Discovery and functional analysis of a novel ALPK1 variant causing ROSAH syndrome

Tom Snelling, L. Olory-Garnotel, Isabelle Jéru…·Alban C. Ziegler

The paper identifies a new ALPK1 variant causing ROSAH syndrome and shows it has altered activation by different metabolites, explaining autoinflammatory episodes.

2 citations

humanexperimental assay

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Genetic & Genomic MedicinemedRxivPreprintJan 28, 2024

Unveiling Endoglin non canonical regulation: spotlight on the new role of the uPAR pathway

Gaëlle Munsch, Carole Proust, Clémence Deiber…·Omar Soukarieh

This study identifies ENG 5UTR variants altering upstream ORFs that reduce Endoglin levels and links uPAR pathway loci to soluble Endoglin levels through proteogenomics and functional assays.

1 citation

humanproteomics

𝕏 PostBluesky