Pamela Altamirano-Silva

This study identifies an actin-dependent mechanism by which Brucella abortus exits host cells in infective clumps, forming extracellular vesicles that can reinfect neighboring cells.

This study identifies three modes of Brucella abortus exit from host cells, including cell-to-cell transmission, which may enhance immune evasion and persistence.

This study shows that CDT toxin from Clostridioides difficile induces biofilm-like microcolonies, increasing colonization and persistence in intestinal models and infected mice.