Mayd M. Alsalman

The paper compares mitochondrial morphology and function in hippocampal CA1 and CA2 circuits, finding cell-type and input-specific differences that may influence circuit properties and disease vulnerability.

This study investigates how murine cerebral capillary endothelial and pericyte cells respond to blood flow cessation, finding that pericytes mediate microvascular constriction via ET-1 and alter endothelial tight junctions.

This study shows that MCU regulates synaptic plasticity specifically at CA2 distal dendrites by maintaining mitochondrial morphology and distribution, with loss of MCU causing LTP deficits and mitochondrial fragmentation.

The paper shows that mitochondria in CA2 distal dendrites are larger and enriched in MCU, linking synaptic activity to mitochondrial morphology and function.