Author

Akmal Salimov

University of PennsylvaniaORCID 0009-0002-0186-3708Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Assay:flow cytometry· 1 Approach:statistical model· 1

Frequent Co-Authors

Martin P CarrolliD· 2 shared A’ishah Bakayoko· 1 shared Andrew Matthews· 1 shared Angelo D’AlessandroiD· 1 shared Ann DevineiD· 1 shared Brian KeithiD· 1 shared Bryan Manning· 1 shared Caner SayginiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

Martin P CarrollORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintAug 4, 2026

Human CEBPA-N AML exhibits enhanced engraftment and a C/EBPα-p30-driven leukemic stem cell program

Philomina Sona Peramangalam, Mayuresh Vishwas Konde, Emin Onur Karakaslar…·John Anto Pulikkan

This study identifies CEBPA-N-AML as having enhanced engraftment and defines a novel LSC signature in CEBPA-N-AML.

DOI 10.64898/2026.08.03.742558

humanflow cytometry

𝕏 PostBluesky

Cancer BiologyLeukemiaPeer-reviewedJul 9, 2025

Chemoresistance of TP53 mutant AML requires the mevalonate byproduct, GGPP, for regulation of ROS and induction of a mitochondria stress response

Sarah J. Skuli, A’ishah Bakayoko, Marisa Kruidenier…·Martin P Carroll

This study identifies GGPP, a mevalonate pathway byproduct, as critical for chemoresistance in TP53mut AML through ROS regulation and mitochondria stress response, suggesting statin-based therapies could be effective.

12 citationstop 2%

humanbulk RNA-seq

𝕏 PostBluesky