Author

Antonio Mollica

University of TorontoORCID 0009-0002-6775-9735Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:experimental assay· 1 Approach:statistical model· 1 Research Focus:variant calling· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Aideen Teeling· 1 shared Anjali Vig· 1 shared Anne‐Claude GingrasiD· 1 shared Benjamin Akman· 1 shared Cassandra J. WongiD· 1 shared Constance L. Smith‐HicksiD· 1 shared Edith Cheng· 1 shared Eric I. CamposiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperGenetic & Genomic Medicine

Aideen Teeling

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2 papers

Recent·Most cited

PRMT1 arginine methylation of MCM4 restricts ssDNA gap formation during DNA replication

Michael W. Ferguson, Cassandra J. Wong, Xiaohan Yang, Edith Cheng, Antonio Mollica, Shahir M. Morcos, Wade H. Dunham, Zhen-Yuan Lin, Eric I. Campos, Anne‐Claude Gingras, Grant W. Brown

This study identifies PRMT1 as a replisome-associated methyltransferase that suppresses ssDNA gap formation by methylating MCM4, thereby maintaining fork integrity during DNA replication.

bioRxiv · Jun 4, 2026 · PreprintMolecular Biology

Brain development mutations in the β-tubulin TUBB result in defective ciliogenesis

Antonio Mollica, Safia Omer, Georgiana Forguson, Sydney Steiman, Sonia L. Evagelou, Serhiy Naumenko, Susan Walker, L Y Li, Aideen Teeling, Kyle Lindsay, Steven Erwood, Shagana Visuvanathan, Anjali Vig, Robert McCoy Vernon, Benjamin Akman, , Evgueni A. Ivakine

The paper identifies a novel TUBB mutation linked to both ciliopathy and tubulinopathy brain features, showing it impairs ciliogenesis by altering microtubule dynamics.

Nature Communications · Nov 27, 2025Peer-reviewedGenetic & Genomic Medicine