Author

Selcan Kahveci-Türköz

RWTH Aachen UniversityORCID 0009-0002-9008-131XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Organism:human· 1 Research Focus:protein design· 1 Assay:protein sequence· 1 Assay:protein structure· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Aaron BabendreyeriD· 2 shared Andreas LudwigiD· 2 shared Stefan DüsterhöftiD· 2 shared C Martin· 1 shared Caroline C. F. GrannemanniD· 1 shared Charlotte Flasshove· 1 shared Christine LuxiD· 1 shared Colin AdrainiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersPharmacology & Toxicology

Aaron BabendreyerORCID

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2 papers

Recent·Most cited

Pharmacology & ToxicologybioRxivPreprintJun 9, 2026

The pseudoproteinase iRhom2 critically promotes acute lung inflammation

Christine Lux, Selcan Kahveci-Türköz, Katharina Schun…·Andreas Ludwig

This study shows that iRhom2 deficiency reduces acute lung inflammation in mice by impairing ADAM17-mediated shedding of inflammatory mediators and neutrophil recruitment.

DOI 10.64898/2026.06.05.730404

mouseexperimental assay

𝕏 PostBluesky

Molecular BiologyCellular and Molecular Life SciencesPeer-reviewedFeb 26, 2024

Pathological mutations reveal the key role of the cytosolic iRhom2 N-terminus for phosphorylation-independent 14-3-3 interaction and ADAM17 binding, stability, and activity.

Katharina Bläsius, Lena Ludwig, Sarah Knapp…·Stefan Düsterhöft

This study identifies a phosphorylation-independent 14-3-3 interaction site in iRhom2's N-terminus that regulates ADAM17 activity and shows how mutations in this region cause different pathological outcomes.

12 citations

humanmouse

𝕏 PostBluesky