Author

Truman Knowles

University of California, San FranciscoORCID 0009-0002-9501-823XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:perturbation response prediction· 1 Approach:wet-lab / biochemical assay· 1

Frequent Co-Authors

Charles Stiles· 1 shared Christopher J. GraseriD· 1 shared Daphne Adele Haas-KoganiD· 1 shared Dipanjan ChowdhuryiD· 1 shared Franziska Michor AiD· 1 shared Graeme Smith· 1 shared Helen Robinson· 1 shared Heng Wang· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Charles Stiles

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Cancer BiologybioRxivPreprintMay 15, 2026

H3K27M-driven hypertranscription leads to a new targetable dependency in diffuse midline gliomas

Sharmistha Pal, Heng Wang, Jacob Geisberg…·Daphne Adele Haas-Kogan

This study identifies hypertranscription as a vulnerability in H3K27M-driven diffuse midline gliomas, showing ATR inhibition effectively targets this feature in preclinical models.

DOI 10.64898/2026.05.12.724628

mousehuman