Author

Fabian Frechen

University of BonnORCID 0009-0003-1279-2842Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Dagmar WachteniD· 2 shared Agnes SchröderiD· 1 shared Andreas Ulrich LindneriD· 1 shared Anil AkbaliD· 1 shared Annika Frauenstein· 1 shared Anushka KudaliyanageiD· 1 shared Benedikt O. GanseniD· 1 shared Carmen Ruiz de AlmodóvariD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCell Biology

Dagmar WachtenORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintFeb 25, 2026

NLRP3 activators disrupt the endocytic AP2 complex and plasma membrane signaling

Stefan Ebner, Kshiti Meera Phulphagar, Yubell Alvarez…·Felix Meissner

The paper shows NLRP3 activators disrupt the AP2 complex and impair plasma membrane signaling, leading to inflammation and cell death.

DOI 10.64898/2026.02.24.707400

humanproteomics

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Cell BiologyThe EMBO JournalPeer-reviewedAug 20, 2025

BBSome-dependent ciliary Hedgehog signaling governs cell fate in the white adipose tissue

Katharina Sieckmann, Nora Winnerling, Dalila Juliana Silva Ribeiro…·Dagmar Wachten

This study shows that primary cilia dysfunction in Bbs8-/- mice reduces adipose stem cells and promotes fibrogenic progenitors via disrupted Hedgehog signaling, affecting adipogenesis and lipid uptake.

10 citations

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