Author

Baijun Dong

Shanghai Jiao Tong UniversityORCID 0009-0003-3192-6949Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Helen He ZhuiD· 2 shared Wei XueiD· 2 shared Ang Li· 1 shared Bo LiuiD· 1 shared Chaping Cheng· 1 shared Haotian Wu· 1 shared Huifang ZhaoiD· 1 shared Jiahua PaniD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

Helen He ZhuORCID

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2 papers

Recent·Most cited

Glucokinase links metabolism and lineage plasticity in neuroendocrine prostate cancer via interaction with AKT1

Kai Shen, Ruopeng Su, Yiyi Ji, W. Zhang, Xinyu Liu, Xinyu Chai, Junyi Wang, Bo Liu, Ang Li, Haotian Wu, T. Wang, Xiang Zhou, Zhou Jiang, Helen He Zhu, Liang Dong, , Wei Xue

This study identifies glucokinase (GCK) as a metabolic enzyme that regulates lineage plasticity in neuroendocrine prostate cancer through its interaction with AKT1.

bioRxiv · Jan 7, 2026 · PreprintCancer Biology

E-cadherin bridges cell polarity and spindle orientation to ensure prostate epithelial integrity and prevent carcinogenesis in vivo

Xue Wang, Kai Zhang, Zhongzhong Ji, Chaping Cheng, Huifang Zhao, Yaru Sheng, Xiaoxia Li, 樊连城, Baijun Dong, Wei Xue, Wei‐Qiang Gao, Helen He Zhu

This study shows that E-cadherin connects cell polarity and spindle orientation in prostate epithelial cells, preventing carcinogenesis.

PLoS Genetics · Aug 17, 2018Peer-reviewedDevelopmental Biology