Author

Brandon T Paradoski

University of ManitobaORCID 0009-0004-7727-906XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Aaron J. MarshalliD· 1 shared Ayesha SaleemiD· 1 shared Danielle Fowke· 1 shared Edgard M. Mejia· 1 shared Folayemi Olayinka‐Adefemi· 1 shared Grant Michael HatchiD· 1 shared Hu ZengiD· 1 shared Nissim HayiD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

Aaron J. MarshallORCID

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1 paper

Recent·Most cited

PI3K-dependant reprogramming of hexokinase isoforms controls glucose metabolism and functional responses of B lymphocytes

Brandon T Paradoski, Sen Hou, Edgard M. Mejia, Folayemi Olayinka‐Adefemi, Danielle Fowke, Grant Michael Hatch, Ayesha Saleem, Versha Banerji, Nissim Hay, Hu Zeng, Aaron J. Marshall

This study shows that PI3K-dependent upregulation of hexokinase 2 (HK2) controls glucose metabolism and immune functions in B cells, with HK2-deficient B cells showing impaired metabolic and functional responses.

iScience · Sep 13, 2024Peer-reviewedImmunology