Author

David I. Karambizi

Brown UniversityORCID 0009-0006-1099-5373Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:ChIP-seq· 2 Approach:experimental assay· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:gene regulatory network inference· 1 Assay:Hi-C· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:spatial domain detection· 1

Frequent Co-Authors

András FiseriD· 2 shared Nikos TapinosiD· 2 shared Adrianne Corseri· 1 shared Daniel Lee· 1 shared Eduardo J. FajardoiD· 1 shared Eduardo J. Fajardo· 1 shared Hye‐Yeon HwangiD· 1 shared Jia‐Shu CheniD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCancer Biology

András FiserORCID

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2 papers

Recent·Most cited

Cancer BiologyJournal of Biological ChemistryPeer-reviewedSep 17, 2025

Inhibition of HDAC7 reprograms the histone H3.3 landscape to induce heterochromatin spreading and DNA replication defects in cancer cells.

Ola Hassan, Mattia D. Pizzagalli, Owen P. Leary…·Nikos Tapinos

This study shows that inhibiting HDAC7 alters H3.3 deposition and chromatin structure, leading to DNA replication defects and increased sensitivity to DNA damage in cancer cells.

5 citations

humanChIP-seq

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GenomicsbioRxivPreprintMay 2, 2022

Role of the EGR2 promoter antisense RNA in modulating chromatin accessibility and spatial genome organization

Margot Martinez-Moreno, David I. Karambizi, Hye‐Yeon Hwang…·Nikos Tapinos

This study shows that the EGR2 promoter antisense RNA modulates chromatin accessibility and spatial genome organization by interacting with histone modification complexes.

DOI 10.1101/2022.05.02.490330

mouseChIP-seq

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