Author

F.B. Gil

Universidad de Los Andes, ChileORCID 0009-0006-1666-7987Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Daniel Paredes‐SabjaiD· 2 shared Marjorie Pizarro‐GuajardoiD· 2 shared Nicolás Montes-BravoiD· 2 shared Pablo Castro-CórdovaiD· 2 shared A. Said Álamos-MusreiD· 1 shared Carolina OteroiD· 1 shared Christian Brito-SilvaiD· 1 shared Diego Cea· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersPharmacology & Toxicology

Daniel Paredes‐SabjaORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Pharmacology & ToxicologybioRxivPreprintOct 1, 2026

Repurposing nystatin and taurocholate to target intracellular spore persistence during antibiotic treatment to prevent recurrent Clostridioides difficile infection.

Pablo Castro-Córdova, Maria Mendoza-Leon, A. Said Álamos-Musre…·Daniel Paredes‐Sabja

This study shows that combining nystatin, taurocholate, and antibiotics, with nanoparticle delivery, reduces recurrent Clostridioides difficile infection by targeting intracellular spores.

DOI 10.64898/2026.09.25.754266

mouseexperimental assay

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MicrobiologybioRxivPreprintSep 25, 2026

Gut complement C1q and C3 interact with Clostridioides difficile spores via CdeM and contribute to pathogenesis.

Marjorie Pizarro‐Guajardo, Christian Brito-Silva, Pablo Castro-Córdova…·Daniel Paredes‐Sabja

This study identifies complement proteins C1q and C3 as interacting with C. difficile spores via CdeM, influencing spore entry, persistence, and recurrence of infection.

DOI 10.64898/2026.09.24.753972

humanproteomics

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