Author

Kenta Hata

Tokyo Medical and Dental UniversityORCID 0009-0007-9025-0271Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:organoid / primary culture· 2

Frequent Co-Authors

Taku SatoiD· 2 shared Toshiaki OhtekiiD· 2 shared Daisuke HishikawaiD· 1 shared Hajime SatoiD· 1 shared Hiroki Yotsumata· 1 shared Hiroshi HaenoiD· 1 shared Hiroyuki HaradaiD· 1 shared Itoshi NikaidoiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCancer Biology

Taku SatoORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJun 18, 2026

Modeling the MRD state reveals the insomnia of chemotherapy-tolerant persister clones

Kenta Hata, Taku Sato, Yuki Fukawa…·Toshiaki Ohteki

The paper models MRD states to identify chemotherapy-tolerant persister clones in tongue cancer, revealing their role in metastatic relapse.

DOI 10.64898/2026.06.14.732194

humanorganoid / primary culture

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJun 10, 2026

Cycling persister clones with elevated NR2F1-mediated cholesterol biosynthesis cause chemotherapy resistance

Hajime Sato, Taku Sato, Yohei Sasagawa…·Toshiaki Ohteki

The paper identifies cycling persisters (CPs) in cancer organoids and shows that elevated NR2F1-mediated cholesterol biosynthesis contributes to chemotherapy resistance.

DOI 10.64898/2026.06.06.730520

humanorganoid / primary culture

𝕏 PostBluesky