Author

Mohammed Kamran Afzal Mirza

Babraham InstituteORCID 0009-0008-2467-4882Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Annette Gabriele Beck‐SickingeriD· 1 shared Anowara Islam· 1 shared David J. HodsoniD· 1 shared Dorottya HorkaiiD· 1 shared Gavin A. BewickiD· 1 shared Hanane Hadj‐MoussaiD· 1 shared James Alexander PearsoniD· 1 shared Jonathan M. HouseleyiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

Annette Gabriele Beck‐SickingerORCID

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2 papers

Recent·Most cited

Decoupling AMPK from fatty acid synthesis allows maintenance of fitness late in life

Hanane Hadj‐Moussa, Megan Ulusan, Dorottya Horkai, Mohammed Kamran Afzal Mirza, Jonathan M. Houseley

This paper shows that activating AMPK without inhibiting fatty acid synthesis preserves late-life fitness in yeast by modulating acetyl-CoA metabolism.

eLife · Jul 31, 2026Peer-reviewedMolecular Biology

Neuropeptide Y4 receptor activation delays autoimmune diabetes by reprogramming β-cell stress and immune tolerance

Naila Haq, Klaudia Toczyska, Anowara Islam, Oladapo Edward Olaniru, Yuxian Lei, Ming Hu, M Zhao, R Müller, Mohammed Kamran Afzal Mirza, Nick Fine, David J. Hodson, Shanta Jean Persaud, Annette Gabriele Beck‐Sickinger, James Alexander Pearson, Gavin A. Bewick

This study shows that activating the Y4R receptor protects β-cells and modulates islet immunity, delaying autoimmune diabetes in multiple models.

bioRxiv · Jul 4, 2026 · PreprintCell Biology