Author

Natsuko HitoraーImamura

Hokkaido UniversityORCID 0009-0009-3227-3904Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:electronic health records· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:spatial domain detection· 1

Frequent Co-Authors

Ayumu KonnoiD· 1 shared Hirokazu HiraiiD· 1 shared Hiroshi KatsukiiD· 1 shared Hiroshi NomuraiD· 1 shared Hiroyuki HiokiiD· 1 shared Kazuki OkamotoiD· 1 shared Kensuke Motomura· 1 shared Kyoka Nishimura· 1 shared

Activity by Year

2018-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperNeuroscience

Ayumu KonnoORCID

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3 papers

Recent·Most cited

NeuroscienceNeuronPeer-reviewedJun 1, 2026

Infraslow histaminergic dynamics govern priming states to gate moment-to-moment memory accessibility

Yoshikazu Morishita, Yuki Takamura, Kyoka Nishimura…·Hiroshi Nomura

The paper shows that infraslow histaminergic activity in mice modulates memory expression by priming BLA circuits, influencing moment-to-moment memory accessibility.

DOI 10.1016/j.neuron.2026.05.019

mouseelectronic health records

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NeuroscienceJournal of Pharmacological SciencesPeer-reviewedApr 24, 2026

Establishment of spinocerebellar ataxia type 34 model mice accompanied by early glial activation and degeneration of cerebellar neurons

Yuri Morikawa-Yujiri, Kensuke Motomura, Ayumu Konno…·Takahiro Seki

This study establishes a SCA34 mouse model with early glial activation and cerebellar neuron degeneration, linking microglial activation to motor dysfunction and neurodegeneration.

DOI 10.1016/j.jphs.2026.04.004

mouseexperimental assay

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NeuroscienceJournal of NeuroimmunologyPeer-reviewedJun 7, 2018

GABAergic malfunction underlying maternal immune activation-induced social deficits

Kazuki Okamoto, Natsuko HitoraーImamura, Hiroyuki Hioki, Yuji Ikegaya

This study identifies GABAergic dysfunction in the anterior cingulate cortex as a mechanism underlying social deficits caused by maternal immune activation in mice.

27 citations

mouseelectronic health records

𝕏 PostBluesky