Abigail G Parrish

This study identifies TEAD inhibition as a therapeutic target in meningioma and shows that combining MEK and mTOR inhibition overcomes resistance in resistant lines.

The paper identifies PTEN loss as a driver of aggressive NF2 mutant meningiomas and validates a cross-species model for preclinical testing.

This study investigates how C-terminal fusion partners, specifically TFE3 domains, contribute to the oncogenic activity of YAP1::TFE3 fusion proteins in tumors.

This study measures cortisol responses to lab stressors in juvenile cichlid fish and finds no behavioral-cortisol associations.

This study shows ERK signaling promotes resistance to TRK inhibitors in NTRK fusion-driven glioma mouse models and suggests MEK inhibition as a potential combination therapy.

The paper shows that aggressive high-grade NF2 mutant meningiomas have reduced YAP1 activity due to increased VGLL4 and FAT3/4 expression, linked to higher recurrence risk.