ROS function as a distinct mitochondrial retrograde response factor for appropriate cardiogenic specifications and cardiac function in Drosophila embryos
Abstract
This manuscript has been withdrawn by the author as it was submitted and made public without the full consent of all the authors. Therefore, the author does not wish this work to be cited as reference for the project. If you have any questions, please contact the corresponding author.
02 /Specification & Classification
Click any tag to browse matching papersReproducibility ledger
Nobody has reported trying to reproduce this paper yet. Did you replicate an experiment or run the code? Your report saves the next lab a week.
Sign in to file a reproducibility report.
Related papers
- Drosophila ryanodine receptor gene triggers functional and developmental muscle properties and could be used to assess the impact of human RYR1 mutations
Monika Zmojdzian, Teresa Jagla, … Catherine Sarret·eLifePeer-reviewed·May 13, 2026
- Deficiency in MICOS component Chchd3 Compromises Drosophila Heart Function via mitophagy, ROS and ER Stress
Cristiana Dondi, Shuchao Ge, … Rolf Bodmer·bioRxiv·Aug 19, 2026
- Mitochondrial Fission Regulates Transcription of Ribosomal Protein Genes in Embryonic Hearts
Qiancong Zhao, Shun Yan, … Kai Jiao·bioRxiv·Feb 10, 2021
- ROS signalling requires uninterrupted electron flow and is lost during ageing in flies
C. Graham, R. Stefanatos, … A. Sanz·bioRxiv·Aug 18, 2021
- EFHD1 ablation reduces cardiac mitoflash activation and protects cardiomyocytes from ischemia.
D. R. Eberhardt, S. H. Lee, … D. Chaudhuri·bioRxiv·Oct 18, 2021
- Mitochondrial Complex I and ROS control neuromuscular function through opposing pre- and postsynaptic mechanisms
Bhagaban Mallik, Sajad Ahmad Bhat, … C. Andrew Frank·PLoS BiologyPeer-reviewed·Sep 22, 2025