Author

A. A. Gad

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:proteomics· 1

Frequent Co-Authors

D. A. Annis· 1 shared D. E. Gilchrist· 1 shared J. A. Ju· 1 shared K. N. Thompson· 1 shared K. T. Chang· 1 shared L. Boyman· 1 shared M. B. Stemberger· 1 shared M. I. Vitolo· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperCancer Biology

D. A. Annis

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Disruption of P2Y2 signaling promotes breast tumor cell dissemination by reducing ATP-dependent calcium elevation and actin localization to cell junctions

M. L. Mull, S. J. P. Pratt, K. N. Thompson, D. A. Annis, A. A. Gad, R. M. Lee, K. T. Chang, M. B. Stemberger, J. A. Ju, D. E. Gilchrist, L. Boyman, M. I. Vitolo, W. J. Lederer, S. S. Martin

This study shows that P2Y2 receptor signaling, which increases intracellular calcium and actin localization in non-tumorigenic breast cells, is disrupted in metastatic cells, promoting dissemination.

bioRxiv · Mar 31, 2023Cancer Biology

Conserved residues in the extracellular loop 2 regulate Stachel-mediated activation of ADGRG2

A. A. Gad, P. Azimzadeh, N. Balenga

This study identifies conserved residues in ECL2 of ADGRG2 that are critical for Stachel-mediated activation and surface expression, revealing their role in signaling and degradation.

bioRxiv · Feb 14, 2021Pharmacology & Toxicology