Author

A. E. Mullick

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:host-pathogen & immune response· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Daugherty· 2 shared D. A. Howatt· 2 shared H. S. Lu· 2 shared H. Sawada· 2 shared J. J. Moorleghen· 2 shared M. K. Franklin· 2 shared A. C. Trumbauer· 1 shared A. Ji· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersPathology

A. Daugherty

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3 papers

Recent·Most cited

Renal Proximal Tubule Cell-specific Megalin Deletion Does Not Affect Atherosclerosis But Induces Tubulointerstitial Nephritis in Mice Fed Western Diet

N. Amioka, M. K. Franklin, M. Kukida, L. Zhu, J. J. Moorleghen, D. A. Howatt, Y. Katsumata, A. E. Mullick, M. Yanagita, M. Martinez-Irizarry, R. M. Sandoval, K. W. Dunn, H. Sawada, A. Daugherty, H. S. Lu

This study shows that deleting megalin in renal proximal tubule cells does not reduce atherosclerosis but causes tubulointerstitial nephritis in male mice fed a Western diet.

bioRxiv · May 11, 2024Pathology

Antisense Oligonucleotide Targeting Hepatic Serum Amyloid A Limits the Progression of Angiotensin II-Induced Abdominal Aortic Aneurysm Formation

P. Shridas, A. Ji, A. C. Trumbauer, V. P. Noffsinger, L. W. Meredith, F. C. de Beer, A. E. Mullick, N. R. Webb, D. G. Karounos, L. R. Tannock

This study shows that reducing SAA with antisense oligonucleotides limits AAA progression in obese mice treated with AngII.

bioRxiv · Aug 22, 2023Pathology

Inhibition of Angiotensin II Dependent AT1a Receptor Stimulation Attenuates Thoracic Aortic Pathology in Fibrillin-1C1041G/+ Mice

J. Z. Chen, H. Sawada, J. J. Moorleghen, M. K. Franklin, D. A. Howatt, M. B. Sheppard, A. E. Mullick, H. S. Lu, A. Daugherty

This study shows that inhibiting the AngII-AT1aR axis reduces aortic pathology in male Fbn1C1041G/+ mice, suggesting ligand-dependent AT1aR stimulation contributes to TAA progression.

bioRxiv · Jun 1, 2020Physiology