Author

A. G. Manjon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

F. M. Feringa· 2 shared J. van den Berg· 2 shared R. H. Medema· 2 shared B. van den Broek· 1 shared K. Jalink· 1 shared K. Kielbassa· 1 shared L. Koob· 1 shared L. Krenning· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCell Biology

F. M. Feringa

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2 papers

Recent·Most cited

DNA end-resection in highly accessible chromatin produces a toxic break

J. van den Berg, S. E. P. Joosten, Y. Kim, A. G. Manjon, L. Krenning, L. Koob, F. M. Feringa, R. Klompmaker, B. van den Broek, K. Jalink, W. Zwart, R. H. Medema

The authors withdrew their manuscript due to inconsistencies in data and questionable experimental results regarding DNA end-resection and chromatin accessibility.

bioRxiv · Aug 30, 2021 · PreprintCell Biology

A limited number of double-strand DNA breaks are sufficient to delay cell cycle progression.

J. van den Berg, A. G. Manjon, K. Kielbassa, F. M. Feringa, R. Freire, R. H. Medema

The paper shows that a single DNA double-strand break can trigger a checkpoint response that delays cell cycle progression, and its abrogation leads to micronucleated daughter cells.

bioRxiv · May 7, 2018Cell Biology