Author

A. Kueh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Research Focus:synaptic & neural circuits· 1

Frequent Co-Authors

A. K. Voss· 1 shared B. Tran· 1 shared C. Hall· 1 shared C. McLean· 1 shared D. Frank· 1 shared D. Komander· 1 shared E. Leong· 1 shared E. Tsui· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCell Biology

A. K. Voss

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Trabid patient mutations impede the axonal trafficking of adenomatous polyposis coli to disrupt neurite growth

D. Frank, M. Bergamasco, M. Mlodzianoski, A. Kueh, E. Tsui, C. Hall, G. Kastrappis, A. K. Voss, C. McLean, M. C. Faux, K. Rogers, B. Tran, E. Vincan, D. Komander, G. Dewson, H. Tran

This study identifies Trabid mutations that disrupt APC trafficking and neurite growth by impairing DUB activity or STRIPAK binding, leading to neurodevelopmental defects.

bioRxiv · Jul 17, 2023Cell Biology

The SOCS1 KIR and SH2 domain are both required for suppression of cytokine signaling in vivo

K. Doggett, N. Keating, F. Dehkhoda, G. M. Bidgood, E. Leong, A. Kueh, N. A. Nicola, N. J. Kershaw, J. J. Babon, W. S. Alexander, S. E. Nicholson

This study shows that both the KIR and SH2 domains of SOCS1 are essential for its function in suppressing cytokine signaling, as mutations in either domain lead to neonatal lethality and inflammation.

bioRxiv · Dec 20, 2022 · PreprintCell Biology