Author

C. A. Eyster

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

K. M. Humphries· 2 shared S. Matsuzaki· 2 shared A. Batushansky· 1 shared A. M. Pendas· 1 shared A. Pranay· 1 shared B. L. Loveland· 1 shared D. S. Dawson· 1 shared E. L. Kurdzo· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

K. M. Humphries

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Loss of cardiac PFKFB2 drives Metabolic, Functional, and Electrophysiological Remodeling in the Heart

K. M. Harold, S. Matsuzaki, A. Pranay, B. L. Loveland, A. Batushansky, M. F. Mendez Garcia, C. A. Eyster, S. Stavrakis, Y. A. Chiao, M. Kinter, K. M. Humphries

This study shows that loss of cardiac PFKFB2 leads to metabolic, functional, and electrophysiological changes in the heart, including impaired systolic function and altered electrophysiology.

bioRxiv · Nov 22, 2023Physiology

Diabetes induced decreases in PKA signaling in cardiomyocytes: the role of insulin

C. A. Eyster, S. Matsuzaki, J. R. Giorgione, K. M. Humphries

This study shows that diabetes impairs PKA signaling in cardiomyocytes, with insulin deficiency contributing to reduced phosphorylation of PFK-2, a key metabolic regulator.

bioRxiv · Apr 2, 2020Cell Biology

Shugoshin protects centromere pairing and promotes segregation of non-exchange partner chromosomes in meiosis

L. Previato, J. M. Evatt, H. H. Chuong, E. L. Kurdzo, C. A. Eyster, M. N. Gladstone, L. Gomez-H, E. Llano, R. Meyer, A. M. Pendas, R. J. Pezza, D. S. Dawson

This paper shows that shugoshin protects centromeric synaptonemal complex components and promotes proper segregation of non-crossover partner chromosomes in meiosis.

Proceedings of the National Academy of Sciences · Apr 24, 2019Peer-reviewedCell Biology