Author

A. Malita

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Animal Behavior & Cognition· 2 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 4 Approach:experimental assay· 4 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell type annotation· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

K. Rewitz· 4 shared M. J. Texada· 4 shared S. Nagy· 3 shared T. Koyama· 3 shared M. Lassen· 2 shared N. Ahrentloev· 2 shared O. Kubrak· 2 shared G. W. Maurer· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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4 shared papersAnimal Behavior & Cognition

K. Rewitz

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

A gut-derived hormone switches dietary preference after mating in Drosophila

A. Malita, O. Kubrak, T. Koyama, N. Ahrentloev, K. V. Halberg, M. J. Texada, S. Nagy, K. Rewitz

This study identifies a gut hormone in Drosophila that shifts dietary preference to protein-rich food after mating to support reproduction.

bioRxiv · Mar 2, 2022 · PreprintAnimal Behavior & Cognition

Insulin signaling couples growth and early maturation to cholesterol intake

M. J. Texada, M. Lassen, L. H. Pedersen, A. Malita, K. Rewitz

This study shows that dietary cholesterol promotes growth and early maturation in Drosophila by enhancing insulin-like peptide expression through glial and fat-tissue relays.

bioRxiv · Jun 7, 2021 · PreprintCell Biology

The gut hormone Allatostatin C regulates food intake and metabolic homeostasis under nutrient stress

O. Kubrak, L. Jensen, N. Ahrentloev, T. Koyama, A. Malita, M. T. Naseem, M. Lassen, S. Nagy, M. J. Texada, K. Halberg, K. Rewitz

bioRxiv · Dec 5, 2020 · PreprintPhysiology

Analysis of genes within the schizophrenia-linked 22q11.2 deletion identifies interaction of night owl/LZTR1 and NF1 in GABAergic sleep control

G. W. Maurer, A. Malita, S. Nagy, T. Koyama, T. Werge, K. A. Halberg, M. J. Texada, K. Rewitz

This study identifies LZTR1 (night owl) and its interaction with Nf1 in regulating GABAergic sleep control, linking them to schizophrenia and 22q11.2 deletion syndrome.

bioRxiv · Oct 23, 2019Animal Behavior & Cognition