Author

A. S. Mason

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 2 Assay:DNA methylation· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. A. Antson· 1 shared A. Alati· 1 shared A. M. Waters· 1 shared A. Macdonald· 1 shared A. Piapi· 1 shared A. S. Woolf· 1 shared D. A. Long· 1 shared D. Coverley· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperCell Biology

A. A. Antson

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Author profile →

3 papers

Recent·Most cited

Dominant CIZ1 fragments drive epigenetic instability and are expressed in early stage cancers

G. Turvey, E. Lopez de Alba, E. Stewart, L. Byrom, H. Cook, S. Sofi, A. Alati, J. Ainscough, A. S. Mason, A. A. Antson, D. Coverley

This study identifies that elevated CIZ1 anchor domain fragments in early stage cancers disrupt SMACs, leading to epigenetic instability by destabilizing PRC1-dependent histone modifications.

bioRxiv · Sep 22, 2023 · PreprintCell Biology

Exploration of the single-cell transcriptomic landscape identifies aberrant glomerular cell crosstalk in a murine model of WT1 kidney disease

J. C. Chandler, D. J. Jafree, S. Malik, G. Pomeranz, M. Ball, M. Kolatsi-Joannou, A. Piapi, W. J. Mason, A. S. Woolf, P. J. Winyard, A. S. Mason, A. M. Waters, D. A. Long

This study uses single-cell RNA sequencing to identify disrupted cell-cell communication in a mouse model of WT1 kidney disease, highlighting non-immunological pathology and suggesting vascular-targeted therapies.

bioRxiv · Oct 11, 2022 · PreprintCell Biology

BK polyomavirus (BKPyV) is a risk factor for bladder cancer through induction of APOBEC3-mediated genomic damage

S. C. Baker, A. S. Mason, R. G. Slip, K. T. Skinner, A. Macdonald, O. Masood, R. S. Harris, T. R. Fenton, M. Periyasamy, S. Ali, J. Southgate

This study shows BKPyV infection increases APOBEC3-mediated genomic damage in urothelial cells, contributing to bladder cancer risk in immunocompromised individuals.

bioRxiv · May 13, 2021Cancer Biology