Author

A. Sanchi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:chromatin & epigenetic regulation· 2 Assay:ATAC-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Organism:yeast· 1

Frequent Co-Authors

M. Lopes· 3 shared J. Krietsch· 2 shared A. Chanou· 1 shared A. Scialdone· 1 shared B. Welter· 1 shared C. Merigliano· 1 shared C. S. K. Lee· 1 shared D. Dibitetto· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersCancer Biology

M. Lopes

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Replication fork plasticity upon replication stress requires rapid nuclear actin polymerization

M. D. Palumbieri, C. Merigliano, D. Gonzalez-Acosta, T. von Kaenel, B. Welter, H. Stoy, J. Krietsch, S. Ulferts, A. Sanchi, R. Grosse, I. Chiolo, M. Lopes

This study shows that nuclear actin polymerization enables replication fork plasticity by facilitating fork slowing and reversal during replication stress.

bioRxiv · Mar 24, 2023 · PreprintCancer Biology

Single molecule MATAC-seq reveals key determinants of DNA replication origin efficiency

A. Chanou, M. Weiss, K. Holler, T. Straub, J. Krietsch, A. Sanchi, H. Ummethum, C. S. K. Lee, E. Kruse, M. Trauner, M. Werner, M. Lalonde, M. Lopes, A. Scialdone, S. Hamperl

The paper develops MATAC-Seq to analyze single-molecule chromatin accessibility at replication origins, identifying chromatin features that predict origin efficiency in yeast.

bioRxiv · Mar 14, 2023Biochemistry

Fork Slowing and Reversal as an Adaptive Response to Chronic ATR Inhibition

D. Dibitetto, A. Sanchi, E. J. Sanford, M. Lopes, M. B. Smolka

This study investigates the effects of chronic low-dose ATR inhibition on replication dynamics, revealing fork reversal as an adaptive response that enhances cell survival and can be exploited to improve cancer therapies.

bioRxiv · May 18, 2021 · PreprintCancer Biology