Author

A. Walaszczyk

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

E. Dookun· 2 shared G. D. Richardson· 2 shared H. Arthur· 2 shared J. Chapman· 2 shared J. F. Passos· 2 shared J. Mialet-Perez· 2 shared S. Tual-Chalot· 2 shared W. A. Owens· 2 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

E. Dookun

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Clearance of senescent cells during cardiac ischemia-reperfusion injury improves heart recovery following myocardial infarction

E. Dookun, A. Walaszczyk, R. Redgrave, P. Palmowski, S. Tual-Chalot, A. Suwana, J. Chapman, E. Jirkovsky, L. Donastorg, E. Gill, Y. Santin, o. E. Yausep, J. Mialet-Perez, W. A. Owens, D. G. Grieve, , G. D. Richardson

This study shows that clearing senescent cells after cardiac ischemia-reperfusion injury improves heart function, reduces scar size, and decreases inflammation.

bioRxiv · Apr 28, 2020 · PreprintCell Biology

Length-independent telomere damage drives cardiomyocyte senescence

R. Anderson, A. Lagnado, D. Maggiorani, A. Walaszczyk, E. Dookun, J. Chapman, J. Birch, H. Salmonowicz, M. Ogrodnik, D. Jurk, C. Proctor, C. Correia-Melo, S. Victorelli, E. Fielder, R. Berlinguer-Palmini, , J. F. Passos

This study shows that telomere damage independent of length causes cardiomyocyte senescence, leading to cardiac dysfunction, and that clearing senescent cells improves cardiac health.

bioRxiv · Aug 17, 2018Cell Biology