Author

R. Berlinguer-Palmini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

C. Albus· 2 shared M. Zorkau· 2 shared A. Lagnado· 1 shared A. Parini· 1 shared A. Walaszczyk· 1 shared C. Correia-Melo· 1 shared C. Hewison· 1 shared C. Proctor· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

C. Albus

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Author profile →

3 papers

Recent·Most cited

Mitochondrial translation occurs preferentially in the peri-nuclear mitochondrial network of cultured human cells.

C. Albus, R. Berlinguer-Palmini, C. Hewison, F. McFarlane, E.-A. Savu, R. Lightowlers, Z. Chrzanowska-Lightowlers, M. Zorkau

The paper shows that mitochondrial translation is preferentially localized in the peri-nuclear mitochondrial network in cultured human cells, despite similar levels of mitoribosomal proteins and rRNA in peripheral regions.

bioRxiv · Sep 15, 2021Cell Biology

High resolution imaging of nascent mitochondrial protein synthesis in cultured human cells.

M. Zorkau, C. Albus, R. Berlinguer-Palmini, Z. M. Chrzanowska-Lightowlers, R. N. Lightowlers

This study uses advanced imaging to show that mitochondrial protein synthesis in human cells mainly occurs at cristae membranes, separate from RNA processing sites.

bioRxiv · May 5, 2020 · PreprintCell Biology

Length-independent telomere damage drives cardiomyocyte senescence

R. Anderson, A. Lagnado, D. Maggiorani, A. Walaszczyk, E. Dookun, J. Chapman, J. Birch, H. Salmonowicz, M. Ogrodnik, D. Jurk, C. Proctor, C. Correia-Melo, S. Victorelli, E. Fielder, R. Berlinguer-Palmini, , J. F. Passos

This study shows that telomere damage independent of length causes cardiomyocyte senescence, leading to cardiac dysfunction, and that clearing senescent cells improves cardiac health.

bioRxiv · Aug 17, 2018Cell Biology