Author

Alexa Rane Batingana

University of California, San Francisco

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

Benjamin J. HuangiD· 3 shared Kevin ShannoniD· 3 shared Jeevitha D’Souza· 2 shared Soheil MeshinchiiD· 2 shared Abigail S. Lin· 1 shared Adam J. LambleiD· 1 shared Aishwarya C. Ballapuram· 1 shared Anica M. WandleriD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersCancer Biology

Benjamin J. HuangORCID

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Author profile →

3 papers

Recent·Most cited

De novo design of Ras isoform selective binders

Jason Z. Zhang, Xinting Li, Alexa Rane Batingana, Caixuan Liu, Hanlun Jiang, Kevin Shannon, Benjamin J. Huang, Kejia Wu, David A. Baker

This study designs Ras isoform-specific binders using deep learning to target the C-termini of Ras proteins, enabling specific inhibition and study of their roles in biology and disease.

Cell chemical biology · Mar 1, 2026Peer-reviewedBioengineering

Acute Myeloid Leukemia Relapse after Bromodomain Inhibitor Treatment or Chemotherapy is Characterized by Myc-Ras Transcriptional Remodeling

Benjamin J. Huang, Jeevitha D’Souza, Alexa Rane Batingana, Max Harris, Xinyue Wang, Eugene Hwang, Anica M. Wandler, Michael R. Burgess, Qing Li, Soheil Meshinchi, Gideon E. Bollag, Kevin Shannon

This study shows that AML relapse after BET inhibitor or chemotherapy involves Myc-Ras transcriptional changes, highlighting transcriptional plasticity in relapsed leukemias.

bioRxiv · Nov 13, 2025 · PreprintCancer Biology

TP53 Inactivation Confers Resistance to the Menin Inhibitor Revumenib in Acute Myeloid Leukemia

Jeevitha D’Souza, Camille J. Leung, Aishwarya C. Ballapuram, Abigail S. Lin, Alexa Rane Batingana, Adam J. Lamble, Rhonda E. Ries, Carolina E. Morales, Charisa Cottonham, Pranav Gona, Li Fan, Xiaotu Ma, Kevin Shannon, Soheil Meshinchi, Benjamin S. Braun, Benjamin J. Huang

This study shows TP53 mutations cause resistance to revumenib in AML and suggests combining MCL-1 inhibitors with menin inhibitors could be effective.

bioRxiv · Mar 24, 2025 · PreprintCancer Biology