Author

Anna Katharina Kuderna

Universität Ulm

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Genomics· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

Thomas StammingeriD· 3 shared Caroline C. FriedeliD· 2 shared Adam T. GrundhoffiD· 1 shared Altuna AkaliniD· 1 shared Anna Heider· 1 shared Anna ReicheliD· 1 shared Anne-Charlotte König· 1 shared Armin EnsseriD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersMicrobiology

Thomas StammingerORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Exonuclease ISG20 inhibits human cytomegalovirus replication by inducing an innate immune defense signature

Matthias Hehl, Myriam Scherer, Cora Stegmann, Eva-Maria Raubuch, Claudia Ploil, Teresa Rummel, Philipp Kirchner, Nina Kottmann, Anna Reichel, Anna Katharina Kuderna, Anne-Charlotte König, Caroline C. Friedel, Florian Erhard, Thomas Stamminger

This study identifies ISG20 as an antiviral factor against HCMV by inducing an innate immune defense signature that enhances IFN production and response.

PLoS Pathogens · Jan 9, 2026Peer-reviewedMicrobiology

Intrinsic immunity against HAdV is achieved by a novel epigenetic silencing complex

Julia Mai, Maryam Karimi, Anna Katharina Kuderna, Anna Heider, Thomas Günther, Caroline C. Friedel, Adam T. Grundhoff, Thomas Stamminger, Sabrina Schreiner

This study identifies the HUSH complex as a host factor that restricts HAdV infection through epigenetic silencing, and shows HAdV proteins inhibit this complex to promote viral replication.

bioRxiv · Feb 10, 2025 · PreprintCell Biology

DNA virus infections shape transposable elements activity in vitro and in vivo

Jiang Tan, Vedran Franke, Eva Neugebauer, Sandra Pennisi, Melanie Anna Schaechtle, Justine Lagisquet, Anna Katharina Kuderna, Stephanie Wälter, Isabelle Welker, Armin Ensser, Markus Landthaler, Thomas Stamminger, Thomas Gramberg, Altuna Akalin, Emanuel Wyler, Florian Full

This study shows DNA virus infections activate transposable elements (TEs) through KAP1/TRIM28 inhibition and DUX4 expression, with in vivo correlations in cancer patients.

bioRxiv · Apr 18, 2024 · PreprintGenomics