Author

B Luther Davies

Centre for Human Genetics

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genomics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Anjali Gupta HinchiD· 1 shared Bradley M. RobertsiD· 1 shared Callum HenfreyiD· 1 shared Daniela MoralliiD· 1 shared Emily H.T. PrintiD· 1 shared Gang ZhangiD· 1 shared Harrison Waters· 1 shared Isabella Maudlin· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperGenomics

Anjali Gupta HinchORCID

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2 papers

Recent·Most cited

A human GBA-L444P transgene drives early and persistent dopamine neurotransmission deficits and alpha-synuclein pathology in a mouse model of early Parkinson's disease

Natalie Connor‐Robson, Tara Diviney, Javier Alegre‐Abarrategui, Bradley M. Roberts, Katherine R. Brimblecombe, Nora Bengoa‐Vergniory, Harrison Waters, Milena Cioroch, B Luther Davies, Kateryna O. Bila, Martijn J. C. van der Lienden, Johannes M. F. G. Aerts, Stephanie Jane Cragg, Richard Wade‐Martins

This paper creates a humanised GBA-L444P mouse model to study GBA1 mutations in Parkinson's, finding gain-of-function effects on dopamine release and loss-of-function effects on alpha-synuclein pathology.

bioRxiv · May 27, 2026 · PreprintNeuroscience

A genome-wide atlas of meiotic recombination intermediates reveals distinct modes of DNA repair that direct crossovers away from transcriptionally marked genes

Callum Henfrey, Emily H.T. Print, Gang Zhang, Robert Hinch, Isabella Maudlin, Daniela Moralli, B Luther Davies, Peter Donnelly, Anjali Gupta Hinch

The paper maps meiotic recombination intermediates in mice to show two repair modes that direct crossovers away from transcriptionally active genes.

bioRxiv · Mar 28, 2026 · PreprintGenomics