Author

B.-J. M. Webb-Robertson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:proteomics· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

E. S. Nakayasu· 2 shared A. Chakraborty· 1 shared A. T. Wright· 1 shared C. M. Anderson· 1 shared C. Muralidharan· 1 shared D. L. Eizirik· 1 shared D. Surguladze· 1 shared E. K. Sims· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersBiochemistry

E. S. Nakayasu

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Author profile →

3 papers

Recent·Most cited

A Composite Biomarker Signature of Type 1 Diabetes Risk Identified via Augmentation of Parallel Multi-Omics Data from a Small Cohort

O. Alcazar, S.-T. Chuang, G. Ren, M. Ogihara, B.-J. M. Webb-Robertson, E. S. Nakayasu, P. Buchwald, M. H. Abdulreda

The paper validates 12 biomarkers from a small T1D cohort using data augmentation, identifying 7 significantly altered markers linked to T1D risk.

bioRxiv · Feb 9, 2024 · PreprintBiochemistry

Inhibition of the Eukaryotic Initiation Factor-2-α Kinase PERK Decreases Risk of Autoimmune Diabetes in Mice

C. Muralidharan, F. Huang, J. R. Enriquez, J. E. Wang, J. B. Nelson, T. Nargis, S. C. May, A. Chakraborty, K. T. Figatner, S. Navitskaya, C. M. Anderson, V. Calvo, D. Surguladze, M. J. Mulvihill, X. Yi, , R. G. Mirmira

This study shows that inhibiting PERK reduces autoimmune diabetes risk in mice by decreasing β cell immunogenicity and improving islet function.

bioRxiv · Oct 6, 2023Cell Biology

Proximity-dependent proteomics of the Chlamydia trachomatis inclusion membrane reveals functional interactions with endoplasmic reticulum exit sites

M. S. Dickinson, L. N. Anderson, B.-J. M. Webb-Robertson, J. R. Hansen, R. D. Smith, A. T. Wright, K. Hybiske

This study uses proximity-dependent biotinylation to identify inclusion membrane proteins of Chlamydia trachomatis and reveals a novel interaction with endoplasmic reticulum exit sites.

bioRxiv · Mar 19, 2018Microbiology