Author

C. Pallasch

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2

Frequent Co-Authors

D. Vorholt· 2 shared M. Krueger· 2 shared R. Neuhaus· 2 shared A. Bansal· 1 shared B. Sackey· 1 shared B. Schumacher· 1 shared D. Bachurski· 1 shared E. Izquierdo· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCell Biology

D. Vorholt

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2 papers

Recent·Most cited

Loss of TP53 mediates suppression of Macrophage Effector Function via Extracellular Vesicles and PDL1 towards Resistance against Chemoimmunotherapy in B-cell malignancies

E. Izquierdo, D. Vorholt, B. Sackey, J. Nolte, S. J. Blakemore, J. Schmitz, V. Barbarino, N. Nickel, D. Bachurski, L. Lobastova, M. Nikolic, M. Michalik, R. Brinker, R. Neuhaus, L. Frenzel, , C. Pallasch

This paper identifies that TP53 loss in B-cell lymphoma leads to resistance against chemoimmunotherapy by suppressing macrophage effector function through PDL1-expressing extracellular vesicles.

bioRxiv · Jun 11, 2020 · PreprintCell Biology

ATM-mediated DNA damage response in macrophages primes phagocytosis and immune checkpoint regulation

A. Bansal, R. Neuhaus, E. Izquierdo-Alvarez, H. Nolte, D. Vorholt, H. Feldkoetter, P. Lohneis, R. Buettner, M. Krueger, C. Pallasch, B. Schumacher

The paper shows that DNA damage in macrophages increases PD-L1 expression and phagocytic activity, suggesting a role in chemotherapy response and immune checkpoint regulation.

bioRxiv · Mar 14, 2020 · PreprintImmunology