Author

C. Pin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Ecology· 1

Signature Assays, Organisms & Methods

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Approach:biophysical modeling· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:ecology & biodiversity· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. A. Mangaonkar· 1 shared A. Buradkar· 1 shared A. Godfrey· 1 shared A. Lynam· 1 shared A. Riggio· 1 shared A. Vedder· 1 shared A. Yurchenko· 1 shared B. Alver· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. A. Mangaonkar

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Mapping threatened Thai bovids provides opportunities for improved conservation outcomes in Asia

W. Horpiencharoen, J. Marshall, A. Lynam, R. Muylaert, R. S. John, D. Ngoprasert, A. Godfrey, A. Riggio, G. A. Gale, E. Ash, F. Bisi, G. Cremonesi, G. R. Clements, M. Yindee, N. M. Shwe, , D. Hayman

The paper maps suitable habitats for five threatened bovids in Thailand and identifies conservation opportunities by analyzing protected area coverage.

bioRxiv · Aug 25, 2023Ecology

The stress-induced protein NUPR1 orchestrates protein translation during ER-stress by interacting with eIF2α

M. T. Borrello, P. Santofimia-Castano, M. Bocchio, C. Pin, J. L. Iovanna

This paper identifies NUPR1's role in ER stress response by interacting with eIF2α to regulate protein translation recovery.

bioRxiv · Feb 18, 2020 · PreprintCell Biology

RAS mutations drive proliferative chronic myelomonocytic leukemia via activation of a novel KMT2A-PLK1 axis

R. M. Carr, D. Vorobyev, T. Lasho, D. L. Marks, E. J. Tolosa, A. Vedder, L. Almada, A. Yurchenko, I. Padioleau, B. Alver, G. Coltro, M. Binder, S. L. Safgren, I. Horn, X. You, , M. M. Patnaik

This study identifies RAS mutations as drivers of proliferative CMML through a novel KMT2A-PLK1 axis, showing PLK1 inhibition can improve disease outcomes in patient-derived xenografts.

bioRxiv · Dec 23, 2019 · PreprintCancer Biology