Author

C. Sanchez-Martin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:metabolism & bioenergetics· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Organism:human· 2 Assay:metabolomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

A. Rasola· 2 shared I. Masgras· 2 shared A. Castegna· 1 shared A. Menga· 1 shared A. Pujol· 1 shared C. Laquatra· 1 shared E. Moroni· 1 shared E. Rial· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersCancer Biology

A. Rasola

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Role of UCP2 in the energy metabolism of the cancer cell line A549

J. Segales, C. Sanchez-Martin, A. Pujol, M. Martin-Ruiz, E. Rial

This study investigates the role of UCP2 in A549 cancer cells, finding that UCP2 silencing reduces respiration without increasing glycolysis and does not alter ATP levels or proton leakage, suggesting UCP2 functions as a C4-metabolite carrier rather than an uncoupling protein.

bioRxiv · Sep 13, 2022 · PreprintBiochemistry

Tumor growth of neurofibromin-deficient cells is driven by decreased respiration and hampered by NAD+ and SIRT3

I. Masgras, G. Cannino, F. Ciscato, C. Sanchez-Martin, M. Pizzi, A. Menga, A. Castegna, A. Rasola

This study shows that neurofibromin loss reduces mitochondrial respiration and NAD+ levels, promoting tumor growth, and that restoring NAD+ or SIRT3 can counteract this effect.

bioRxiv · May 29, 2021Cancer Biology

HIF1a-dependent induction of the mitochondrial chaperone TRAP1 regulates bioenergetic adaptations to hypoxia

A. Rasola, C. Laquatra, C. Sanchez-Martin, G. Minervini, E. Moroni, M. Schiavone, S. C. E. Tosatto, F. Argenton, G. Colombo, P. Bernardi, I. Masgras

This study identifies TRAP1 as a HIF1a-dependent regulator of mitochondrial bioenergetics in hypoxic conditions, showing its role in maintaining respiration in developing embryos and cancer models.

bioRxiv · Sep 23, 2020Biochemistry