Author

Claire H. M. Tang

University of Birmingham

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Organism:human· 1

Frequent Co-Authors

Aditi KanhereiD· 2 shared Claire WilsoniD· 2 shared Eva PetermanniD· 2 shared Richard D.W. KellyiD· 2 shared Rosanna J. WilkinsiD· 2 shared Abirami Kannan· 1 shared Martin A.M. ReijnsiD· 1 shared Panagiotis KotsantisiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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2 shared papersCancer Biology

Aditi KanhereORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMar 18, 2026

PI3K-AKT activation determines oncogenic RAS-induced hypertranscription and replication stress

Richard D.W. Kelly, Claire Wilson, Claire H. M. Tang…·Eva Petermann

This study shows that PI3K-AKT activation is required for RAS-induced replication stress and hypertranscription in cancer cells.

1 citation

experimental assaycancer & tumor microenvironment

𝕏 PostBluesky

Molecular BiologyOncogenePeer-reviewedJul 10, 2025

Human RNase H2 upregulation counteracts oncogene- and chemotherapy-induced replication stress

Rosanna J. Wilkins, Abirami Kannan, Siobhan Plass…·Eva Petermann

This study shows that RNase H2 upregulation mitigates replication stress caused by oncogenes and chemotherapy, reducing replication fork stalling and RNA:DNA hybrids.

5 citations

humanexperimental assay

𝕏 PostBluesky