Author

D. F. Jarosz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Evolutionary Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:yeast· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Carbajal· 1 shared C. Jakobson· 1 shared F. McCarty· 1 shared I. Ziv· 1 shared J. Aguilar-Rodriguez· 1 shared J. E. Elias· 1 shared J. E. Snyder· 1 shared J. M. Skotheim· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperEvolutionary Biology

A. Carbajal

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Hsp90 shapes adaptation by controlling the fitness consequences of regulatory variation

C. Jakobson, J. Aguilar-Rodriguez, D. F. Jarosz

This study identifies over 1,000 genotype-to-phenotype associations governed by Hsp90 in yeast, showing cis-regulatory variants have a stronger impact on heredity than coding variation.

bioRxiv · Oct 30, 2023 · PreprintEvolutionary Biology

Genome dilution by cell growth drives starvation-like proteome remodeling in mammalian and yeast cells

M. C. Lanz, S. Zhang, M. P. Swaffer, L. Hernandez Götz, F. McCarty, I. Ziv, D. F. Jarosz, J. E. Elias, J. M. Skotheim

The paper shows that increasing cell size leads to proteome remodeling similar to starvation, driven by genome dilution in mammalian and yeast cells.

bioRxiv · Oct 16, 2023Cell Biology

The hunt for ancient prions: Archaeal prion-like domains form amyloids and substitute for yeast prion domains

T. Zajkowski, M. D. Lee, S. S. Mondal, A. Carbajal, R. Dec, P. D. Brennock, R. W. Piast, J. E. Snyder, N. B. Bense, W. Dzwolak, D. F. Jarosz, L. J. Rothschild

This paper identifies prion-like domains in Archaea that form amyloids and transmit non-Mendelian inheritance, suggesting prions existed in the last universal common ancestor.

bioRxiv · Jul 20, 2020 · PreprintEvolutionary Biology