Author

D. I. Andersson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Evolutionary Biology· 3 Microbiology· 3

Signature Assays, Organisms & Methods

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Organism:E. coli· 4 Approach:experimental assay· 3 Approach:statistical model· 3 Research Focus:drug response / drug discovery· 2 Organism:bacteria (other)· 1 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

B. A. Berryhill· 2 shared T. Gil-Gil· 2 shared A. M. Babina· 1 shared A. P. Smith· 1 shared B. Kacar· 1 shared B. R. R. Levin· 1 shared C. I. M. Koumans· 1 shared D. Hughes· 1 shared

Activity by Year

2016-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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2 shared papersMicrobiology

B. A. Berryhill

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

SPARK: Systematic Profiling of Antibiotic-Phage Response Kinetics

B. A. Berryhill, T. Gil-Gil, D. L. Sanchez-Hevia, D. I. Andersson, Nikos Fatsis-Kavalopoulos

SPARK quantifies phage-antibiotic interactions using growth curves and shows synergy, antagonism, and dynamic changes across bacterial isolates and time.

bioRxiv · Nov 9, 2026 · PreprintMicrobiology

Suppression of the E. coli rnpA49 conditionally lethal phenotype via different compensatory mutations

A. M. Babina, L. A. Kirsebom, D. I. Andersson

The paper identifies temperature-resistant suppressor mutations in E. coli that compensate for the rnpA49 mutation in RNase P, revealing roles for Lon protease and RNase R in the mutant's degradation.

bioRxiv · Dec 5, 2023 · PreprintEvolutionary Biology

Theoretical Considerations and Empirical Predictions of the Pharmaco- and Population Dynamics of Heteroresistance

B. R. R. Levin, B. A. Berryhill, T. Gil-Gil, J. A. Manuel, A. P. Smith, J. E. Choby, D. I. Andersson, D. S. Weiss, F. Baquero

This paper develops two mathematical models of heteroresistance to explore how resistance ascends and reverts during antibiotic treatment, showing that current identification methods are insufficient.

bioRxiv · Sep 21, 2023Microbiology

The chemotherapeutic drug methotrexate selects for antibiotic resistance

J. S. Guthmundsdottir, E. G. A. Fredheim, C. I. M. Koumans, J. Hegstad, P.-C. Tang, D. I. Andersson, O. Samuelsen, P. J. Johnsen

This study shows methotrexate can select for antibiotic resistance genes in E. coli when linked to trimethoprim resistance, at sub-inhibitory concentrations.

bioRxiv · Nov 12, 2020Microbiology

Predicting trajectories and mechanisms of antibiotic resistance evolution

F. Pinheiro, O. Warsi, D. I. Andersson, M. Lässig

The paper develops a fitness model to predict antibiotic resistance evolution by linking metabolic effects to growth rates and resistance levels in E. coli.

bioRxiv · Jul 2, 2020 · PreprintEvolutionary Biology

Functional constraints on replacing an essential gene with its ancient and modern homologs

B. Kacar, E. Garmendia, N. Tuncbag, D. I. Andersson, D. Hughes

This study replaces the essential tufA gene in E. coli with extant and ancestral homologs, showing that functional transfer depends on the presence of tufB and evolutionary distance.

bioRxiv · Nov 15, 2016Evolutionary Biology