Author

D. M. Dickey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Cataliotti· 1 shared A. J. Hobbs· 1 shared B. M. Noel· 1 shared C. Navis· 1 shared C. Perez-Ternero· 1 shared D. A. Bernlohr· 1 shared F. O. Levy· 1 shared H. Andresen· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPharmacology & Toxicology

A. Cataliotti

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2 papers

Recent·Most cited

Novel enhancers of guanylyl cyclase-A activity via allosteric modulation

H. Andresen, C. Perez-Ternero, J. Robinson, D. M. Dickey, A. J. Hobbs, L. R. Potter, F. O. Levy, A. Cataliotti, L. R. Moltzau

The paper identifies small molecule allosteric enhancers of GC-A that increase ANP and BNP signaling without using known binding sites or mechanisms.

bioRxiv · Dec 31, 2021Pharmacology & Toxicology

Multi-omic profiling of tyrosine kinase inhibitor-resistant K562 cells suggests metabolic reprogramming to promote cell survival

B. M. Noel, S. B. Ouellette, L. Marholz, D. M. Dickey, C. Navis, T.-Y. Yang, V. Nguyen, S. J. Parker, D. A. Bernlohr, Z. Sachs, L. L. Parker

This study identifies metabolic reprogramming and novel markers in TKI-resistant CML cells through multi-omic profiling.

bioRxiv · Jan 16, 2019 · PreprintCancer Biology